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Elevated circulatory cortisol levels, resulting from enhanced HPA axis activity due to depression, may represent another possible biological mechanism that triggers CHD ( Indeed, depression can lead to changes in neuroendocrine function, activating both the HPA axis and the sympathetic nervous system

A later RNA i screen of a library of 11,511 genes determined that the silencing of a total of 37 C

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10.1126/science.aaf5171 178 MayersJ